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After initial results, many researchers maintain lower frequency protocols rather than stopping entirely
Other neurotoxicity mechanisms have also been suggested, including the METH-induced increase in reactive oxidative stress and ROS levels, which activate myosin light chain protein kinase, thereby reducing TJ protein expression (Zhang et al., 2017)
The medication maintains consistent blood levels regardless of cycle phase, meaning dose timing and efficacy remain predictable
doi:10.1007/s13300-024-01674-8
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