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Israel et al., A number of studies have shown that ASA, even at low anticlotting doses, acutely acetylates cyclooxygenase-1 (Cox-1) in platelets, and chronically, it also acetylates Cox-2 leading to the generation of a powerful anti-inflammatory agent
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(3) equipping the transmeningeal drug delivery device with a sealing mechanism to achieve site-specific effects
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