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Although studies on the relationship between HO-1 and macrophages in diabetes or its complications are limited, the mechanisms by which macrophages maintain the microenvironment stability of the body and HO-1s excellent antioxidant and anti-inflammatory properties suggest that targeting HO-1 with macrophages may be a treatment choice for diabetes (Fig
379 Therapeutic modulation of the autophagy-mitophagy axis in NDDs Pharmacological agents Multiple pharmacological agents enhance autophagy through distinct regulatory nodes
Myricetin supplementation decreases hepatic lipid synthesis and inflammation by modulating gut microbiota
However, not all glutathione supplements are created equal
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