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However, N-acetyl-p-benzoquinone imine (NAPQI) approximately 5% of a therapeutic dose is metabolized by the liver enzyme cytochrome P450 2E1 to the electrophile N-acetyl-p-benzoquinone imine (NAPQI)

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In this article, we will review incretin physiology and evidence supporting a role of the incretin hormones in the pathophysiology of hyperinsulinemic hypoglycemia, as well as results from proof-of concept studies exploring a therapeutic approach targeting the GLP-1 receptor to treat hyperinsulinemic hypoglycemia

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