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In the early stages of the disease, bacterial metabolic products in dental plaque can activate host cells to release inflammatory mediators like interleukin-1, tumor necrosis factor-, further contributing to the release of oxygen free radicals and other types of oxidants (Mazurek-Mochol et al., 2024)

This study noted that animals pretreated with CRE and CRE-AgNPs responded with better resumption in the activity of GST than irradiated animals not treated with the C

Each enzyme targets specific macronutrients, breaking them down more efficiently to maximize nutrient absorption and reduce digestive discomfort

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