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This constitutive abundance combined with injury-site concentration creates a research context in which exogenous TB-500 is supplementing and amplifying an endogenous repair biology rather than introducing a foreign signal a distinction with implications for how TB-500s research findings are interpreted in the context of endogenous tissue repair mechanisms

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Circulation 100, II361II364

"Prevention of Bone Loss and Management of Fracture Risk in HIV-Infected Individuals: Case Studies and Recommendations for Different Patient Subgroups." Future Virol 6.6 (2011): 769-782