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The released GHK-Cu promoted bone marrow stem cell proliferation, induced neovascularization, and facilitated macrophage polarization toward the M2 (anti-inflammatory, pro-repair) phenotype
CD38 Dictates Age-Related NAD Decline and Mitochondrial Dysfunction through an SIRT3-Dependent Mechanism
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This study was supported by grants from the Hainan Province Science and Technology Special Fund (ZDYF2025LCLH012), the National Key R&D Program of China (2023YFC2306800), the Key Research and Development Project of Zhejiang Province (2024C03149 and 2023C03046), the National Natural Science Foundation of China (82470690 and 92159202), the Fundamental Research Funds for the Central Universities (2025ZFJH03)
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