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Peptides occupy a central position in AD pathobiology: pathogenic species such as A oligomers and tau-derived fragments drive synaptic failure, mitochondrial dysfunction, and neuroinflammation, whereas several endogenous neuropeptides exert compensatory neuroprotective, trophic, or homeostatic effects

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Avoid repeated freeze-thaw cycles, which reduce recovery of the 43-residue thymosin beta 4 chain in particular, label every aliquot with the reconstitution date, and handle throughout under sterile laboratory conditions

[1] [2] It also contains many serous glands, which produce thin saliva