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Once in the nucleus, it binds to specific transcription factors and DNA elements particularly antioxidant response elements (ARE) that are regulated by the stress-responsive factor NRF2. Through this mechanism, MOTS-c can turn on a suite of stress-defense and metabolic genes that help cells adapt to high-fat stress or insulin resistance
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Probably so that he can put me on the operating table
At the same time, it slows down the rate at which food leaves the stomach and reduces appetite by acting on appetite centers in the brain
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