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Intranasal administration of Ex-4 at a dose of 0.5 g/kg daily for 7 days before MCAO in mice, rather than intraperitoneal administration at an equivalent dose, exerted neuroprotective effect by reducing infarct volume and neurological deficits which were blocked by knocking down GLP-1 receptor with shRNA (136)
Sensitization of TRPV1 by EP1 and IP reveals peripheral nociceptive mechanism of prostaglandins
Even more striking, among patients who did develop acute pancreatitis, GLP-1 RA users had fewer complications (including reduced rates of sepsis, AKI, shock, mechanical ventilation) and lower all-cause mortality
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