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Neither drug is universally better, effectiveness varies based on individual tolerance and overall health status
Other mechanisms The administration of ICIs may lead to the generation of autoantibodies that target antigens expressed by podocytes, mesangial cells, or TECs [178]
In microglia, GLP-1R activation via PI3K/Akt signaling also reduces neuroinflammation by modulating NF-B activity, decreases the production of pro-inflammatory cytokines, increases anti-inflammatory markers such as IL-10, IL-4, Arginase, and CD206, and the transformation of microglia into an anti-inflammatory state
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