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It supports the breakdown and transport of fats for more efficient use
It selectively promotes insulin secretion by pancreatic -cells and inhibits glucagon secretion by pancreatic -cells under hyperglycemic conditions, steadily regulating fasting and postprandial blood glucose
Conversion to NADH sustains mitochondrial electron transport, whereas NADPH supports reductive biosynthesis and glutathione-dependent antioxidant buffering [83]
placebo on the time to occurrence of a composite cardiovascular outcome (nonfatal myocardial infarction, nonfatal stroke, cardiovascular death, hospitalization or urgent visit due to heart failure) and a composite renal outcome (end-stage kidney disease, 40% sustained decline in eGFR, cardiovascular death or renal death) in 10,000 individuals with atherosclerotic cardiovascular disease and/or CKD
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