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Based on reading some of the websites it seems that their hypothesis is that because ALC is involved in the Krebs cycle in the mitochondria, which gets energy from fatty acids, that means ALC helps burn fat. There is not much in the scientific literature on this topic, but I did come across an article that found no difference in metabolic parameters in study participants taking L-carnitine versus controls (not exactly the same things as acetyl-L-carnitine, but they are used somewhat similarly in the body, there just isnt the extra acetyl group in the L-carnitine)
These medications enhance satiety, slow gastric emptying, and improve glycemic control, leading to spontaneous reduction in food consumption
Despite years of confusion around the relative benefits of GIPR agonism versus antagonism, the clinical picture that both can enhance the weight-lowering efficacy of a GLP-1R agonist seems clear
That pattern simply does not exist in the data
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