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Understanding the connection between MTHFR and longevity requires moving past the standard homocysteine narrative to the full mechanistic picture: how reduced methylation capacity accelerates the epigenetic aging clock, how it depletes the glutathione substrate pool, how it uncouples eNOS and accelerates vascular aging, how it impairs DNA repair through uracil misincorporation, and how it reduces the methylation-dependent gene regulation that FOXO3 and SIRT1 require to execute their longevity programs
BPC-157 and PRP are increasingly used together in regenerative medicine, and the logic is sound
Glutathione is a naturally produced antioxidant found throughout the body
it is only used within formally approved clinical trials and is not available on NHS prescription
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