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APE1, as a DNA repair enzyme, has the ability to regulate epithelial ROS by regulating the interaction of Rac1 and Nox1, which, in turn, inhibits the production of NADPH oxidase, thus reducing the level of ROS (den Hartog et al., 2016)
The compound exhibited notable cytotoxicity in cells with elevated DT-diaphorase expression and enabled the simultaneous monitoring of the in vitro drug activation process
Hematologic Cancers Among Patients with Type 2 Diabetes Prescribed GLP-1 Receptor Agonists
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