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The same logic applies here
Specific binding of GPR174 by endogenous lysophosphatidylserine leads to high constitutive G(s) signaling
However, it may increase the chance of AKI in indirect ways, especially in people who are already at risk
However, under some metabolic conditions (e.g., reperfusion of anaerobic tissues) unpaired electrons gain access to molecular oxygen in unregulated, non-enzymatic reactions
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