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While therapeutic strategies aimed at enhancing cardiac contractility through the upregulation of SERCA2a or AC6 expression show promise for ameliorating HF symptoms, this approach faces significant limitations
The molecular basis of the pharmacological resistance are still poorly understood, and several potential molecular mechanisms have been proposed, including defective expression or genetic alterations of DRD2 and SSTRs, or an impaired signal transduction
Comput Hum Behav
Lens differentiation is controlled by the balance between PDGF and FGF signaling
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