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Oxid Med Cell Longev 2013, 972913.CrossRefGoogle ScholarPubMed
FOXO4-DRI's ability to selectively eliminate these cells provides researchers with tools for studying how senescent cell burden contributes to aging phenotypes and age-related diseases
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This may involve targeting the CBX3/NRF2/GPX2 axis with CBX3 or NRF2 inhibitors or impairing antioxidant buffering systems such as glutathione synthesis (e.g., via GCLC suppression)
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