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Generally, the toxic material basis of TCM-induced liver injury can be classified into alkaloids, glycosides, anthraquinones, terpenoids lactones, phytotoxic proteins and heavy metals [10] (Table 1)
Its higher fat content and tougher digestion make it a less suitable option
(54) found that administering GIPR antagonist to healthy humans reduced postprandial insulin secretion and increased glucose excursions more than administering GLP-1R antagonist (GLP-1RA), indicating that endogenous GIP may play a more significant role in stimulating insulin secretion and reducing postprandial plasma glucose excursions compared to endogenous GLP-1
& Dringen, R
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