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Our results revealed, the peripapillary nerve fiber layer thickness measured in all quadrants superiorly, temporally, inferiorly and nasally for the three groups using 3D- OCT 2000 showed no significant difference statistically among groups

This observation is consistent with the established model of acetaminophen nephrotoxicity, wherein the toxic metabolite NAPQI depletes GSH, the cell's primary antioxidant defense system, leading to an accumulation of ROS, mitochondrial damage, and subsequent cellular dysfunction (4, 5)

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However, GCL is upregulated by estradiol, leading to a higher concentration of glutamyl-cysteine (Glut-Cys) in females, thereby driving increased GS flux