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USD27.87 USD64.87

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Description

It facilitated an increase in Treg levels and a decrease in IL-17 secretion in an NOD mouse model

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Mitophagy constitutes a critical adaptive survival mechanism for tumor cells under therapeutic pressure by eliminating treatment-induced damaged mitochondria, limiting reactive oxygen species accumulation, and maintaining mitochondrial homeostasis, thereby providing a buffering capacity for the development of resistance

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