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Because nicotinamide would otherwise re-enter the NAD+ salvage pathway through NAMPT and NMN, NNMT activity sits at a junction between cellular methylation capacity and NAD+ regeneration, which is why the enzyme became a target after Kraus and colleagues (Nature, 2014) reported the consequences of antisense Nnmt knockdown in white adipose tissue and liver
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W., Kim, M.-S., Park, J.-Y., Park, K.-G., Lee, H.-J., Lee, I.-K., Lim, S., Jang, H
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