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Mechanistically, SGLT2 inhibitors act predominantly through hemodynamic and cardiorenal pathways, whereas GLP-1 receptor agonists exert metabolic, anti-inflammatory, and anti-atherosclerotic effects, providing a rationale for potential complementarity
Both animal and human studies demonstrate that LPS stimulate GLP-1 release via Toll-like receptors (TLRs)
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Research on irradiation-exposed animals showed Epithalon inhibits apoptotic death of spleen lymphocytes, suggesting protective effects on immune cell populations during stress conditions
