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In individuals with obesity, the brain's reward centres may respond more intensely to food cues, whilst satiety signals may become blunted
They may result in using medication to ease symptoms beyond prescribed limits
These results indicate that the increased amount of non-12-OH FXR-antagonistic BAs consequent to the decrease in CYP8B1 expression after T3 treatment potentiates GLP-1 production by deactivating the intestinal FXR, thereby improving glucose homeostasis via the augmentation of insulin secretion
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