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Luo M, Zheng L-W, Wang Y-S, Huang J-C, Yang Z-Q, Yue Z-P, et al

decreased smooth muscle cell proliferation reduces neointimal formation Anti-inflammatory signaling: Suppression of NF-kB, reduced macrophage infiltration into plaques, and decreased secretion of IL-6, TNF-alpha, and MCP-1 from monocytes and foam cells Atherogenic lipid reduction: Improvements in LDL cholesterol, VLDL, triglycerides, and chylomicron remnants beyond what weight loss alone would predict Blood pressure reduction: Through direct vasodilation and natriuresis, independent of weight change Epicardial and perivascular fat reduction: This fat depot exerts direct paracrine inflammatory effects on the vascular endothelium and myocardium

In general, if someone is struggling to meet their nutrition needs while on a GLP-1 inhibitor, stopping the prescription may be necessary

A similar size spectrum is, of course, well recognized for pulmonary neoplasms