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Both showed benefit, but results were not broken down by medication

[6] Preclinical studies in animal models suggest that GLP-1 receptor activation may: Promote neuroplasticity and neurogenesis (formation of new neurons) in the hippocampus, a region often showing reduced volume in depression Modulate neurotransmitter systems , particularly dopamine and serotonin pathways involved in mood regulation and reward processing Reduce neuroinflammation , which is increasingly recognised as contributing to depressive disorders Enhance brain-derived neurotrophic factor (BDNF) expression, a protein crucial for neuronal health and resilience Metabolic improvements associated with GLP-1 therapy may also contribute indirectly to mood benefits

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Comparison to Other GLP-1 Medications Retatrutide's BP reduction exceeds other obesity medications: Semaglutide (Wegovy): -6.2 mmHg systolic Tirzepatide (Zepbound): -8.0 mmHg systolic (15mg, SURMOUNT-1) Retatrutide: -14.0 mmHg systolic The addition of glucagon receptor agonism likely contributes