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Direct innervation of the PB by neurons originating in the NTS and AP in the brainstem and in the hypothalamic ARH have been demonstrated (88, 89), suggesting an activation pathway for endogenous blood-borne hormones, such as leptin (27), ghrelin (90), and GLP-1 (8), that affects PB signaling
Semaglutide has recently received expanded regulatory approval for use in patients with T2DM and CKD, reflecting growing confidence in its renal safety profile and potential kidney-protective benefits (26, 27)
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