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The link with lipid metabolism has been provided by employing a siRNA for OCTN2, which lowered carnitine uptake triggering an increased formation of lipid droplets and a decreased cell proliferation

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The modifications made to human GLP-1 include: Amino acid substitutions that resist DPP-4 enzyme degradation Attachment of fatty acid chains (acylation) as seen in semaglutide and liraglutide Fusion with immunoglobulin fragments (as in dulaglutide) to increase molecular size These changes are designed using rational drug design principles and implemented through biotechnology

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