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The implication for practice is that incretin therapy is not a substitute for ApoB-lowering therapy in established ASCVD but is complementary: aggressive ApoB lowering addresses the lipid driver of atherogenesis, while incretin therapy addresses residual inflammatory, vascular, and metabolic risk
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Down-Regulation of microRNA-98 Promoted Apoptosis of TNF- Stimulated Human Fibroblast-Like Synoviocytes via Up-Regulating IL-10
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