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Our study demonstrates that cysteine-deficiency-induced CoA loss results in metabolic reprogramming and inefficiency, leading to urinary excretion of both glycolytic and tricarboxylic-acid-cycle intermediates, ketone bodies and other biosynthetic intermediates, contributing to the observed weight loss
Running distance, food intake and body weight were measured daily before dosing of experimental compounds
Convenient, cost-effective, optimal ratio
These animal findings may help explain split timing in informal protocols, but they cannot set a safe dosing frequency or predict subcutaneous timing in people
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