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La medicina personalizada permitira identificar a los pacientes con mayores dficits antioxidantes o disfuncin mitocondrial mediante biomarcadores especficos, optimizando as la seleccin del tratamiento ms adecuado (por ejemplo, NAC frente a CRWP1 o GSH liposomal)
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Further evidence that deletion of GSTM1 may specifically increase the risk of these histological subtypes was observed by Spurdle et al
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