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Since mitochondria are essential to our very existence, it is unsurprising that the effects of mitochondrial diseases can be severe, and sometimes lethal, spanning a wide-ranging spectrum of physiological and biochemical symptoms [1,3,4] (see Table 1 for examples)

A SQUAMOSA MADS box gene involved in the regulation of anthocyanin accumulation in bilberry fruits

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The review also highlights that the existing body of evidence is heterogeneous due to differences in endpoint definitions, measurement methods, populations, drugs, doses, and follow-up periods