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Although the pathogenesis is not completely clear, the role of mucosal epithelial cell apoptosis, oxidative damage, retention of decomposed products, changes to the mast cells and other factors has aroused attention
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Diabetic kidney lesions of GIPRdn transgenic mice: podocyte hypertrophy and thickening of the GBM precede glomerular hypertrophy and glomerulosclerosis
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