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Published September 9, 2021

In a 2001 study published in Endocrinology , Heffernan and colleagues reported that chronic AOD-9604 administration reduced body weight gain, decreased adipose tissue mass, and increased fat oxidation in obese mice without altering IGF-1 levels or glucose metabolism effects that were abolished in beta3 adrenergic receptor (3-AR) knockout mice, implicating 3-AR signaling as the mechanism of action [1]

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Instead, AOD 9604 is used to study downstream metabolic processes, particularly adipose tissue signaling and lipid metabolism, without activating the broader endocrine cascade associated with the GH axis