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that removal is not an approval
methylphenidate) in ADHD, we hypothesize that the disruption of D2R-DAT protein complex might be a new treatment means for ADHD by enhancing the dopaminergic neurotransmission
M.WilmoreD
Pre-clinical rodent models of tendon rupture have shown combined BPC-157 and TB-500 protocols to produce faster return of tensile strength and superior histological organisation compared to either single-peptide treatment or untreated controls
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