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In HT patients, the induction of neutrophil extracellular traps (NET) increases, which depends on the accumulation of ROS through the activation of nicotinamide adenine dinucleotide phosphate (NADPH) oxidase and the production of interleukin (IL) IL-6 (30)
It shows promise in accelerating wound healing, repairing damaged muscle tissue, and potentially mitigating the effects of cardiovascular disease by promoting cardiac muscle regeneration
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