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GPIIb/IIIa inhibitors (e.g., tirofiban) improve platelet redox balance by blocking integrin-mediated signaling, preventing ROS amplification from NOX2 and thiol oxidation during aggregation ( 4.2.2 Antioxidants Oxidative stress can induce mitochondrial damage and is a key contributor to thrombotic events in diabetes ( 1 (CoQ1) reduced the mtROS and improved the M in platelet mitochondria (Sacks et al., 2021)

A logical extension of this framework is the consideration of another key regulatory system -the gut microbiota, which also makes a substantial contribution to diabetes pathogenesis and interacts with the islet apparatus within the framework of the gutpancreas axis

We often advise researchers to look at the bigger picture when designing these complex protocols

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