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With respect to fibrosis, in vitro evidence suggests that NK cells may exert anti-fibrotic effects by eliminating activated HSCs through NKp46/NCR1-dependent, RAE1/NKG2D-dependent, and TRAIL-dependent mechanisms (110, 111) ( Figure 5A )
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Bacterial challenges generally induced multiple GSTs, however individual members of the GST family were specifically modified depending upon the virulence of bacterial strains and the outcome of interaction
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