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Oxygen and glucose deprivation (OGD) model and cell viability To imitate the in vivo environment of ischemia and hypoxia after fat transplantation, ADSCs were cultured under hypoxic and glucose-free conditions to induce an OGD model
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The results suggested that TB-500 not only potentially enhanced the viability, angiogenesis, and migratory ability of HUVEC but possibly also promoted the expression of angiopoietin-2 (Ang2), TEK receptor tyrosine kinase 2 (tie2), vascular endothelial growth factor A (VEGFA), NOTCH1 intracellular domain (N1ICD), Notch receptor 3 (Notch3), NF-B, and phosphorylated (p)-p65 in HUVEC
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