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We suggest glutathione is one such endogenous source, as further impairing glutamate liberation from the cycle diminishes synaptic activity induced by MeAIB, while increasing glutamate availability can rescues the impairment by MeAIB

Combinations can yield mixed results by individual

It is also worth noting that one of the possible mutations appeared to have a much stronger association with schizophrenia in male populations as opposed to female, probably due to the neuroprotective properties of estrogen

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