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Briefly, these studies revealed that DSePA treatment (at concentrations which are non-toxic for normal cells) inhibited the growth of lung cancer cells by causing reductive stress (marked by the increase in the levels of GSH and GPx) followed by mitochondrial and endoplasmic reticulum-dependent apoptosis [92]
Lipotropics enhance this process by supporting the methylation reactions required to convert fat into transportable forms and by preventing hepatic lipid accumulation that can slow metabolic throughput
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