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FASEB Journal , 25(1), 358-369
Then researchers proposed that heterozygous knockout mice appeared to be a good animal model for individuals homozygous for the C667T polymorphism, whereas the homozygous null mice were a better one for severely MTHFR-deficient individuals 19
Its N-terminal trans-3-hexenoic acid modification makes it more resistant to enzymatic breakdown than sermorelin's GHRH(1-29) fragment, giving it a longer half-life
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