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These findings provided external support for TCMR-associated T-cell metabolic transcriptional remodeling, although the limited number of validation biopsies precluded definitive statistical validation

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Microglial NF-B activation under GSH-depleted conditions creates a pro-inflammatory loop where increased ROS promotes NF-B activation, which upregulates inflammatory cytokines and further stimulates oxidative stress (Liu et al., 2017