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Treatment of bEnd.3 monolayers with 100 ng/ml to 10 g/ml of AOM did not increase monolayer permeability as assessed by diffusion of 10 kDa FITC-dextran across the transwell (Figure 2a)
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In this regard, we have successively documented that the formation of a Bcl-2/p38MAPK heterocomplex into the cytoplasm is the genuine responsible for the inhibition of vRNPs nuclear export
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