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The addition of glucagon-receptor activity to the dual-incretin mechanism (already represented by compounds such as tirzepatide) is hypothesized in the literature to extend the molecule's effects on hepatic lipid handling and energy expenditure beyond what is observed with GLP-1/GIP co-agonism alone
Combining both agents could theoretically amplify appetite suppression and gastrointestinal effects, though long-term safety data on this specific combination is lacking
Thats the premise of Dr
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