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GLP-1 is mainly produced in the ileum and the colon and it is known as the most potent incretin in the human body, which delays gastric emptying and lowers postprandial blood glucose level through augmentation of glucose-dependent insulin release (Lim et al
Considerable between-individual and within-individual variability was observed for oral semaglutide bioavailability, which is usual for molecules with low bioavailability [22]
That's not a worst-case scenario - it's a documented pattern in the weight-loss literature
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