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The study showed that the drug interacted with the GLP-1 receptor to a much higher degree than with the glucagon receptor, indicating that any pharmacological effects were likely mediated via GLP-1R activation
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STEP 4 randomised people whod lost 10.6% on semaglutide to either continue the medication or switch to placebo
That is at odds with a much larger systematic review published in The BMJ , formerly known as the British Medical Journal
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