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In vitro and in vivo treatment of JMF4073 eliminates WT-Bcr-Abl-32D CML cells
Key molecular mechanisms of ACs anti-inflammatory activity are generally through NF-B inhibition, where AC inhibits the phosphorylation and degradation of IB-, thus preventing the nuclear translocation of NF-B p65
doi: 10.3389/fcimb.2021.634981
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