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showed that DEX increased the expression levels of SLC7A11 and GPX4 and protected H9C2 cells from hypoxia/reoxygenation injury through the AMPK/GSK-3/Nrf2 axis [233]
Engineering drugs that resist microbial enzymatic degradation, such as CDD-L and resistant gemcitabine analogs, can circumvent intratumoral drug inactivation
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