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LPS-mediated NFB activation and pro-inflammatory cytokine production decreased in neutrophil NFB led to a weakened inflammatory response in the acute phase of mice with acute lung injury, suggesting that H 2 O 2 can inhibit the acute excessive inflammatory response (Lubos et al., 2010)
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Cagrilintide targets the amylin receptor, whereas GLP-1 agonists target glucagon-like peptide-1 pathways
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